Frank Müller, Institute of Structural Biology and Biophysics, Cellular Biophysics (ISB-1) and University of Aachen
Selected Publications
Gotzes, S., de Vente, J. und Müller, F. (1998): Nitric oxide modulates cGMP levels in neurons of the inner and outer retina in opposite ways. Visual Neuroscience 15:945-955.
Müller, F., Bönigk, W., Sesti, F. und Frings, S. (1998): Phosphorylation within a regulatory domain of mammalian olfactory cyclic nucleotide-gated channels increases ligand sensitivity. Journal of Neuroscience 18:164-173.
Körschen, H.G., Beyermann, M., Müller, F., Heck, M., Vantler, M., Koch, K.W., Kellner, R., Wolfrum, U., Bode, C., Hofmann, K.P. und Kaupp, U.B. (1999): Interaction of glutamic-acid-rich proteins with the cGMP signalling pathway in rod photoreceptors. Nature 400:761-766.
Müller, F., Vantler, M., Weitz, D., Eismann, E., Zoche, M., Koch, K.W. und Kaupp, U.B. (2001): Ligand sensitivity of the α2 subunit from bovine cone cGMP-gated channel is modulated by protein kinase C but not by calmodulin. Journal of Physiology 532: 399-409.
Stevens, D.R., Seifert, R., Bufe, B., Müller, F., Kremmer, E., Gauss, R., Meyerhof, W., Kaupp, U.B. und Lindemann, B. (2001): The hyperpolarization-activated channels HCN1 and 4 mediate reponses to sour stimuli. Nature 413(6856):631-635.
Müller, F., Scholten, A., Ivanova, E., Haverkamp, S., Kremmer, E., und Kaupp, U.B. (2003): HCN channels are differentially expressed in retinal bipolar cells and concentrated at synaptic terminals. European Journal of Neuroscience 17: 2084-2096.
Ivanova, E. und Müller, F. (2006): Retinal bipolar cells differ in their inventory of ion channels. Visual Neuroscience 23: 143-154.
Mataruga, A., Kremmer, E. und Müller, F. (2007): Type 3a and 3b OFF-cone bipolar cells provide for the alternative rod pathway in the mouse retina. Journal of Comparative Neurology 502: 1123-1137.
Knop, G., Seeliger, M.W., Thiel, F., Mataruga, A., Kaupp, U.B., Friedburg, C., Tanimoto, N, und Müller, F. (2008): Light responses in the mouse retina are prolonged upon targeted deletion of the HCN1 channel gene. European Journal of Neurosience 28:2221-2230